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Androgens, whether in excess (Hyperandrogenism) or in deficiency (Hypoandrogenism), cause oxidative stress by affecting immunological responses, fostering chronic inflammation, causing mitochondrial dysfunction, and causing oxidative damage in reproductive organs, thereby affecting fertility and reproductive health ( Table 1 )

Impairment of -glucosidase, acid trehalase, trehalose-6-phosphate synthase and class II fructose diphosphate aldolase confers antifungal activities to some inhibitors

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While human outcome data for hair regrowth is less robust than the skin literature, the mechanism is plausible and supported by the broader body of tissue-repair research